Abstract

Priming CD8 T cells against pathogens, tumors, and vaccinations results largely from cross-presentation of exogenous antigens by type 1 conventional dendritic cells (cDC1s). While cDC2 and monocyte-derived cells can cross-present in vitro, their physiological relevance remains unclear. Here, we used genetic models to define the distinct roles of cDC subsets in presentation of various antigen forms in vivo: tumor antigens, immune complexes, and vaccines comprised of mRNA and lipid nanoparticles (mRNA-LNP). For tumor antigen, cDC1s were necessary and sufficient to prime both CD4 and CD8 T cells. In contrast, for immune complexed antigen, either cDC1 or cDC2, but not monocyte-derived cells, could prime CD8 T cells via a shared WDFY4-dependent pathway. Notably, mRNA-LNP vaccinations elicit potent CD8 T cell responses independently of cDC1, WDFY4, and even MHC-I expression on APCs. Instead, cDC2s are a prominent component driving mRNA-LNP-mediated CD8 T cell responses and engage cross-dressing of peptide-MHC-I complexes from non-hematopoietic cells. Single cell analyses further revealed the phenotypic differences in CD8 T cells primed by cDC1 versus cDC2, suggesting their possible functional discrepancies between mRNA-LNP vaccinations and natural infections.

Committee Chair

Kenneth Murphy

Committee Members

Kodi Ravichandran; Megan Cooper; Takeshi Egawa; Wayne Yokoyama

Degree

Doctor of Philosophy (PhD)

Author's Department

Biology & Biomedical Sciences (Immunology)

Author's School

Graduate School of Arts and Sciences

Document Type

Dissertation

Date of Award

12-18-2025

Language

English (en)

Author's ORCID

0000-0003-0217-4808

Available for download on Thursday, December 17, 2026

Included in

Biology Commons

Share

COinS