Abstract

The causes of neurodegenerative disorders have long been of interest, and recent findings have pointed to these disorders beginning outside of the brain in other parts of the body. Connections between the brain and the gut have been postulated as a possible way in which amyloid aggregation observed in neurodegeneration can begin or occur. In this work, we aim to study the idea of cross seeding between bacterial amyloid proteins and human amyloid proteins in a yeast model to explore the phenotypes, properties and cross seeding capabilities of these proteins in-vivo. A background on neurodegeneration, bacterial amyloids and cross seeding is first presented to give understanding on amyloids and nature of the cross-seeding hypothesis. Next, the development and/or use of yeast model systems for the study of CsgB and PSM bacterial amyloid proteins from Escherichia coli and Staphylococcus aureus respectively. The toxicity of the proteins as well as their aggregated phenotype in the yeast is discussed for CsgB first, followed by testing it in yeast models expressing neurodegenerative yeast proteins. After establishing its possible colocalization capabilities alongside ��-synuclein, the same experiments were repeated with PSM�� and PSMβ proteins, showing increased toxicity in the presence of neurodegenerative proteins as well as aggregated puncta that co-localized alongside ��-synuclein. Overall, the work shown demonstrates the potential that yeast models have as a higher-throughput method for the study of bacterial amyloid proteins in-vivo.

Committee Chair

Professor Timothy Wencewicz

Committee Members

Professor Meredith Jackrel (Advisor) Professor Courtney Reichhardt

Degree

Master of Arts (AM/MA)

Author's Department

Chemistry

Author's School

Graduate School of Arts and Sciences

Document Type

Thesis

Date of Award

Winter 12-14-2024

Language

English (en)

Author's ORCID

https://orcid.org/0000-0002-3697-0565

Available for download on Saturday, April 27, 2052

Included in

Chemistry Commons

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